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cardiovascular disease PhD Projects, Programs & Scholarships

We have 95 cardiovascular disease PhD Projects, Programs & Scholarships

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  The phenotypic and functional identification of angiogenic T-cells for potential use to treat cardiovascular disease Project ID SAS0043
  Dr M Ross, Dr G Wright
Application Deadline: 31 May 2020

Funding Type

PhD Type

PROJECT DESCRIPTION. This project is based within the School of Applied Sciences at Edinburgh Napier University and is led by Dr.
  Modelling the association between blood pressure variability and cardiovascular disease
  Dr J Barrett
Application Deadline: 7 January 2020

Funding Type

PhD Type

Cardiovascular disease is a leading cause of mortality in the UK, with over 150,000 cardiovascular-related deaths each year. By identifying novel cardiovascular risk factors we can improve cardiovascular risk prediction, so that individuals at greatest risk may benefit from preventative intervention.
  Understanding the functional role of the non-coding genome in cardiovascular disease
  Prof N Bobola, Prof N Hanley
Applications accepted all year round

Funding Type

PhD Type

Cardiovascular disease is the leading cause of death worldwide. Inherited DNA sequence variants contribute to human diseases and susceptibility.
  (MRC DTP) Decoding the role of vascular smooth muscle cell heterogeneity in cardiovascular disease by single cell genomics
  Prof N Bobola, Prof N Hanley
Application Deadline: 15 November 2019

Funding Type

PhD Type

Cardiovascular disease is the leading cause of death worldwide. Vascular smooth muscle cells (VSMC) are implicated in the pathogenesis of several types of vascular diseases, such as aneurysm, atherosclerosis, vascular calcification and congenital vascular disease.
  Novel approaches for treating cardiovascular disease
  Dr J Erickson
Applications accepted all year round

Funding Type

PhD Type

Cardiovascular disease is the primary cause of death throughout most of the world, and existing therapeutic options are unsuitable for a large percentage of patients.
  The molecular mechanisms underpinning individual variation in platelet function and their effect on the progression and treatment of cardiovascular disease.
  Dr CI Jones
Applications accepted all year round

Funding Type

PhD Type

Platelets are small blood cells that play a vital role in the chronic and acute progression of Cardiovascular Disease (CVD). Platelets respond to a range of agonists, which are released when blood vessels are damaged, by aggregating together to form thrombi.
  Investigating the molecular mechanisms and consequences of ANP aggregation for cardiovascular disease; a computational and experimental approach
  Dr J Madine, Dr D J Rigden, Dr M Winn
Applications accepted all year round

Funding Type

PhD Type

Atrial natriuretic peptide (ANP) is a cardiovascular hormone synthesised and secreted by myocytes in the atrial wall where it regulates atrial blood volume and vasodilation.
  Common severe childhood infections, innate inflammatory responses and cardiometabolic risk: The VASCular changes aFter INfectious Diseases (VASCFIND) study
  Prof D Burgner, Dr S Bekkering
Applications accepted all year round

Funding Type

PhD Type

SUMMARY. Infection, the commonest reason for childhood hospital admission, is a major driver of inflammation and is associated with cardiometabolic risk and disease.
  Is disease risk conferred during childhood set in stone? Using genetics to understand how modifiable lifestyle changes can help prevent disease in later life
  Dr T Richardson
Application Deadline: 25 November 2019

Funding Type

PhD Type

The University of Bristol is offering a 3.5 year full time PhD in research around Population Health to start in 2019. This studentship is funded through GW4 BioMed MRC Doctoral Training Partnership.
  The molecular mechanisms of thrombosis and haemostasis
  Dr C.E. Hughes
Applications accepted all year round

Funding Type

PhD Type

A healthy cardiovascular system relies on the cells lining the blood vessels (endothelial cells) and heart cells (cardiomyocytes) to function normally.
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