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cell migration PhD Projects, Programs & Scholarships

We have 69 cell migration PhD Projects, Programs & Scholarships

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  Microtubule motors in cell migration and integrin trafficking
  Prof V Allan, Prof P Woodman, Dr P Caswell
Applications accepted all year round

Funding Type

PhD Type

Cell migration is vital for the development and health of multicellular organisms. However, enhanced cell migration is also a hallmark of cancer cells, and contributes greatly to metastasis.
  Understanding microtubule-actin crosstalk in neuronal cell migration
  Prof A M Fry, Prof I D Forsythe
Application Deadline: 7 June 2020

Funding Type

PhD Type

Summary. This BBSRC-funded 4-year PhD studentship will address the mechanism of action of a microtubule-associated protein, EML4, in promoting microtubule-actin crosstalk and neuronal cell migration [1].
  Investigate a novel, unexplored link between WNT signalling and regulators of cell migration in cancer progression using CRISPR-Cas9 and advanced live cell imaging.
  Research Group: Randall Division of Cell and Molecular Biophysics
  Dr M Krause, Dr K Liu
Applications accepted all year round

Funding Type

PhD Type

Cancer is a devastating disease. more than one in three people in the UK will develop cancer in their lifetime. WNT signalling is a key pathway controlling morphogenesis in embryos and it has been shown that perturbations of this pathway promote cancer progression.
  Investigating the role of the protease ADAMTS5 in ovarian cancer
  Dr E Rainero
Applications accepted all year round

Funding Type

PhD Type

Ovarian cancer is the most lethal gynaecological malignancy. Mostly because of late stage diagnosis, the 5-year survival rate is.
  Role of lipids in regulating in vivo chemotaxis
  Dr A Renault
Applications accepted all year round

Funding Type

PhD Type

Aberrant cell migration is a critical factor in the progression of many diseases including cancer and atherosclerosis.
  Investigating the communication between cancer cells and cells in the tumour microenvironment to improve our understanding of epigenetic regulatory mechanisms and identify new targets for cancer therapy
  Prof K Gaston, Dr P-S Jayaraman
Applications accepted all year round

Funding Type

PhD Type

It is well-established that the molecular mechanisms controlling gene expression are disrupted in cancer cells and work in this area has laid the foundations for targeted cancer therapies.
  Adhesion Pathways in Extracellular Matrix Organisation
  Prof J Adams
Applications accepted all year round

Funding Type

PhD Type

The extracellular matrix (ECM) provides structural and mechanical support to tissues and modulates cell phenotype and behaviour in health and disease.
  Identifying mechanisms regulating collective migration of embryonic progenitor cells
  Dr M Smutny, Prof M Balasubramanian, Dr D Köster
Application Deadline: 7 June 2020

Funding Type

PhD Type

Collective cell migration plays a fundamental role during development in the embryo and wound healing in adults, and is also a hallmark of invasive cancer cells [1].
  Does fatty acid uptake by oral cancer cells drive metastasis?
  Dr C Murdoch, Dr H Colley
Applications accepted all year round

Funding Type

PhD Type

The cell surface receptor CD36 belongs to a large scavenger receptor family that primarily bind lipid-containing molecules. Until recently their expression was thought to be restricted to macrophages and endothelial cells but recent data suggest that epithelial cancer cells may also express this receptor.
  Unraveling the signal regulation network of cell adhesion, migration, and invasion in podosomes and invadopodia
  Dr C Yu
Applications accepted all year round

Funding Type

PhD Type

Integrin-mediated adhesions, such as podosomes and invadopodia, are the biophysical anchor to sense and to translate mechanobiological signals into various cellular events.
  The functional characterization of the tumour suppressor gene CSMD1 in breast cancer
  Dr S Bell
Applications accepted all year round

Funding Type

PhD Type

CUB and Sushi multiple domains protein 1 (CSMD1) maps to 8p23, a region deleted in many cancers including breast cancer. Our previous work has established that CSMD1 is an independent prognostic marker in ductal breast cancer, with reduced expression associated with high tumour grade and poor survival1.
  Cutting off the fuel supply to calcium pumps in pancreatic cancer: a novel therapeutic strategy
  Dr J Bruce, Prof K Williams, Dr A P Gilmore
Applications accepted all year round

Funding Type

PhD Type

Background and Rationale. Pancreatic ductal adenocarcinoma (PDAC) has the poorest survival and limited treatment options. Therefore, the search for novel therapeutic targets and drugs designed to selectively kill PDAC cells must remain a central research strategy.
  A functional proteomic approach to reveal how receptor tyrosine kinases trafficking specifies cancer cell responses
  Dr C Francavilla, Prof S Hubbard
Applications accepted all year round

Funding Type

PhD Type

Background and goals. Cells respond appropriately to their surroundings by activating plasma membrane spanning receptors like Receptor Tyrosine Kinases (RTKs) and thereby adapting long-term outputs (e.g.
  New strategies in integrin antagonism
  Research Group: Institute of Cancer Therapeutics
  Dr H Sheldrake
Applications accepted all year round

Funding Type

PhD Type

The integrins are a family of transmembrane receptors which mediate cell-cell and cell-ECM adhesion, and signalling across the cell membrane involved in pathways controlling cell migration, proliferation, differentiation, cell survival and apoptosis.
  Cell biology and evolution of cancer metastasis
  Prof P R Dash
Applications accepted all year round

Funding Type

PhD Type

We are interested in the spread of cancer, a process called metastasis. Metastasis is responsible for the majority of cancer deaths and there are currently no effective treatments to stop this process.
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