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integrin PhD Projects, Programs & Scholarships

We have 5 integrin PhD Projects, Programs & Scholarships

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  Mechanistic mathematical / computational modelling of integrin signalling
  Dr N Savage, Dr M Morgan
Applications accepted all year round

Funding Type

PhD Type

Regulation of adhesion dynamics and mechanosensation controls cell migration, microenvironment remodelling and global cell fate decisions.
  New strategies in integrin antagonism
  Research Group: Institute of Cancer Therapeutics
  Dr H Sheldrake
Applications accepted all year round

Funding Type

PhD Type

The integrins are a family of transmembrane receptors which mediate cell-cell and cell-ECM adhesion, and signalling across the cell membrane involved in pathways controlling cell migration, proliferation, differentiation, cell survival and apoptosis.
  MASTERS BY RESEARCH PROJECT: The underlying mechanism by which hypoxia regulates platelet hyperactivity
  Dr I Hers
Applications accepted all year round

Funding Type

PhD Type

Start 23 September 2019. This MSc by Research is a laboratory-based project which offers the exciting opportunity to spend 1 year in one of our State-of-the-Art laboratories (plus 1 year thesis writing) at the University of Bristol and experience life as a researcher.
  Cell biology and evolution of cancer metastasis
  Dr P R Dash
Applications accepted all year round

Funding Type

PhD Type

We are interested in the spread of cancer, a process called metastasis. Metastasis is responsible for the majority of cancer deaths and there are currently no effective treatments to stop this process.
  The molecular mechanisms underpinning individual variation in platelet function and their effect on the progression and treatment of cardiovascular disease.
  Dr CI Jones
Applications accepted all year round

Funding Type

PhD Type

Platelets are small blood cells that play a vital role in the chronic and acute progression of Cardiovascular Disease (CVD). Platelets respond to a range of agonists, which are released when blood vessels are damaged, by aggregating together to form thrombi.
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