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mouse PhD Projects, Programs & Scholarships

We have 163 mouse PhD Projects, Programs & Scholarships

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  G protein-coupled receptor (GPCR) signalling in the mouse and human gastrointestinal (GI) tract.
  Prof H Cox
Application Deadline: 11 January 2019
To establish the mechanisms of GPR35 signalling in the mouse and human GI tract. GPR35 is an orphan receptor that lacks an endogenous ligand but is highly expressed in mammalian GI tract, particularly in the colon and is implicated in the onset of inflammatory bowel disease (IBD).
  MRC Precision Medicine DTP: Defining phenotypic diversity of endometriosis-associated macrophages and signals responsible for their recruitment and differentiation
  Dr E Greaves, Dr C Vallejos, Dr S Jenkins, Prof A Horne
Application Deadline: 7 January 2019
Background. Endometriosis affects 176 million women worldwide and is associated with debilitating pelvic pain and/or infertility.
  Investigation of the anti-inflammatory effect of curcumin in a mouse model of chronic neuroinflammation – a route for a cure for Alzheimer’s disease?
  Dr E Gyengesi, Prof GM Muench
Applications accepted all year round
This project will investigate the effect of chronic glial activation on brain structure and function, and test the efficacy of two cytokine-suppressive anti-inflammatory drugs (CSAIDs) against chronic glial activation and the resulting neuronal damage.
  Marie Skłodowska-Curie ITN, Early Stage Researcher (iPLACENTA, ESR11): “Enhancing imaging of the placenta in murine models of preeclampsia and standardisation of preeclampsia mouse models”
  Prof A Ahmed
Application Deadline: 14 December 2018
Aston Medical School is looking for an Early Stage Researcher (ESR) to work on the project “Enhancing imaging of the placenta in murine models of preeclampsia and standardisation of preeclampsia mouse models (ESR11)” as part of iPLACENTA network collaborating with 14 other ESRs based in 11 European academic, industrial and clinical institutes.
  MRC Precision Medicine DTP: Investigating modifier transcripts in mouse models of motor neuron disease
  Dr L Murray, Dr E Hudlund
Application Deadline: 7 January 2019
The term “motor neuron disease” encompasses a spectrum of disorders in which motor neurons are the primary pathological target.
  The ecology of infection and immunity in a wild mouse system
  Research Group: Institute of Evolutionary Biology
  Dr A Pedersen, Dr S Babayan
Application Deadline: 13 December 2018
Despite great concern about the current global health threat of worms (helminths) in humans and domestic animals, we still don’t have a clear understanding about how ecological heterogeneity determines burdens, disease, or how to successfully control infections in variable populations.
  Defining the metabolic determinants of intestinal stem cell homeostasis
  Dr A Rufini, Prof F Giorgini
Application Deadline: 6 January 2019
The intestine is the fastest renewing organ. This ability is enabled by the presence of fast proliferating intestinal stem cells (ISCs) that reside in the crypts of Lieberkühn at the base of the intestinal villi.
  Clinical Genetics: Building the skull – normal and abnormal development
  Prof A Wilkie, Dr S Twigg
Application Deadline: 11 January 2019
Working closely with the craniofacial teams based in Oxford and other UK units, we specialise in the application of whole exome and genome sequencing to children born with a serious malformation of the skull termed craniosynostosis.
  Genetics of inherited cardiovascular disease: Using molecular genetic analysis of cardiovascular disease as a tool to define disease mechanisms and therapeutic targets.
  Prof H Watkins, Prof H Ashrafian, Prof C Redwood, Prof M Farrall
Application Deadline: 11 January 2019
Using molecular genetic analysis of cardiovascular disease as a tool to define disease mechanisms and therapeutic targets.
  Fusogenic liposomes: the innovative delivery of compounds into human platelets to reduce animal use in platelet research
  Dr A Pollitt, Prof J M Gibbins
Application Deadline: 7 January 2019
Platelets are small cells in the blood which, when activated, play a critical role in the prevention of excessive bleeding at sites of injury.
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