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"ovarian" AND "cancer"×
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ovarian cancer PhD Projects, Programs & Scholarships

We have 14 ovarian cancer PhD Projects, Programs & Scholarships

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Showing 1 to 10 of 14
  Activation of targeted DNA Damage Response as a novel therapy for Ovarian Cancer
Peter MacCallum Cancer Centre Sir Peter MacCallum Department of Oncology, University of Melbourne
  Dr E. Sanij, Prof R. Pearson
Applications accepted all year round
High-grade serous ovarian cancer (HGSOC) is the most common and aggressive subtype of ovarian cancer and accounts for 70% of all ovarian cancer deaths.
Details
Type/Funding
  Therapy-induced senescence and stemness in ovarian cancer
Peter MacCallum Cancer Centre Sir Peter MacCallum Department of Oncology, University of Melbourne
  Prof R. Pearson, Dr K Chan
Applications accepted all year round
Cellular senescence is a stress response characterized by a robust cell cycle arrest and is a brake for malignant transformation.
Details
Type/Funding
  Targeting the Achilles’ heel – dissecting drug resistance in ovarian cancer
University of Dundee School of Medicine
  Dr G Smith, Dr M Ferguson
Applications accepted all year round
Ovarian cancer, the most common cause of gynaecological cancer death, is frequently diagnosed when the disease is already advanced, when surgery is not always possible.
Details
Type/Funding
  Identification and functional characterisation of BRIT1/MCPH1 synthetic lethal genes to treat breast and ovarian cancer
University of Leeds Faculty of Medicine and Health
  Dr S Bell, Prof C A Johnson
Applications accepted all year round
Women who have undergone surgery for breast and ovarian cancer often have additional chemotherapy to kill residual cancer cells and prevent recurrence.
Details
Type/Funding
  Characterize the role of Treacle (TCOF1) in AKT-regulated ribosome biogenesis
Peter MacCallum Cancer Centre Sir Peter MacCallum Department of Oncology, University of Melbourne
  Prof R. Pearson, Dr J Kang
Applications accepted all year round
Ribosomes are essential for cell growth and proliferation, and their biogenesis requires exquisite regulation.
Details
Type/Funding
  (A*STAR) Can non-coding variants explain missing heritability in familial cancer?
University of Manchester Faculty of Biology, Medicine and Health
  Prof B Newman, Prof G Evans, Dr M Smith
Application Deadline: 31 January 2019
Heterozygous germline variants in a number of tumour suppressor genes cause a spectrum of familial cancers, including early onset breast-ovarian cancer, and colorectal cancer.
Details
Type/Funding
  Application of data science to inherited genetic variants of apoptosis genes and susceptibility to breast cancer
University of Sheffield Oncology and Metabolism
  Prof A Cox, Dr K Walters
Application Deadline: 23 January 2019
Background. The identification of the breast and ovarian cancer genes BRCA1 and BRCA2 two decades ago has resulted in both improved patient risk assessment and the use of PARP-inhibitors for treatment.
Details
Type/Funding
  Inhibition of PRMT5 as a cancer therapy
Peter MacCallum Cancer Centre Sir Peter MacCallum Department of Oncology, University of Melbourne
  Dr K. Sheppard, Prof G. McArthur
Applications accepted all year round
Targeted therapy has had profound impact on outcomes for cancer patients.
Details
Type/Funding
  Parallels between wound healing and cancer – role of epithelial to mesenchymal transition (EMT)
University of Bradford Faculty of Life Sciences
  Research Group: Centre for Skin Sciences
  Dr M J Thornton, Prof A M Graham
Applications accepted all year round
Wound healing and cancer metastasis share common early signalling pathways that have led to the hypothesis that tumours are “wounds that do not heal”, a phrase first coined by Virchow in the 19th century.
Details
Type/Funding
  MRC DiMeN Doctoral Training Partnership: Application of data science to inherited genetic variants of apoptosis genes and susceptibility to breast cancer
University of Sheffield MRC DiMeN Doctoral Training Partnership
  Prof A Cox, Dr A Droop, Dr K Walters
Application Deadline: 21 January 2019
The identification of the breast and ovarian cancer genes BRCA1 and BRCA2 two decades ago has resulted in both improved risk assessment for patients and the use of PARP-inhibitors for treatment.
Details
Type/Funding
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