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reprogramming PhD Projects, Programs & Scholarships

We have 24 reprogramming PhD Projects, Programs & Scholarships

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  Metabolic reprogramming and chemotherapy resistance in triple-negative breast cancer
Peter MacCallum Cancer Centre Sir Peter MacCallum Department of Oncology, University of Melbourne
  Dr K Brown
Applications accepted all year round
Treatment options for the triple-negative breast cancer (TNBC) subtype of breast cancer are limited to conventional chemotherapy agents.
Details
Type/Funding
  Fishing for metabolic clues: Role of the Hippo/Yap pathway in reprogramming metabolism in liver cancer
Peter MacCallum Cancer Centre Sir Peter MacCallum Department of Oncology, University of Melbourne
  Dr A. Cox
Applications accepted all year round
The Hippo/Yap pathway is an evolutionarily conserved cascade that plays a fundamental role in governing organ size control, stem cell homeostasis and cancer.
Details
Type/Funding
  Cutting off the fuel supply to starve cancer: Identifying metabolic vulnerabilities in cancer
Peter MacCallum Cancer Centre Sir Peter MacCallum Department of Oncology, University of Melbourne
  Dr K Brown, Dr A. Cox
Applications accepted all year round
A universal characteristic of all cancer cells is the reprogramming of cell metabolism to provide the energy and building blocks necessary to support proliferation and survival.
Details
Type/Funding
  Metabolic Reprogramming in Liver Cancer: delineating the role of reactive oxygen species (ROS) in tumour development
Peter MacCallum Cancer Centre Sir Peter MacCallum Department of Oncology, University of Melbourne
  Prof T. Tiganis
Applications accepted all year round
The Tiganis Laboratory is seeking a highly enthusiastic student to conduct research in cancer metabolism and obesity-associated liver cancer.
Details
Type/Funding
  FOXA1 mutations in Lobular Breast Cancer: Reprogramming Responses to Tamoxifen
University of Nottingham School of Pharmacy
  Prof D Heery, Dr S Johnston
Applications accepted all year round
Tumours of the breast are often hormone-dependent, meaning their growth is driven by steroid hormones such as estrogen. This hormone acts upon the estrogen receptor (ER), which together with other transcription factors such as FOXA1 drives tumour growth.
Details
Type/Funding
  Defining the role of mitochondrial dysfunction in therapy resistance inKRAS mutant colorectal cancer models
Queen’s University Belfast School of Medicine, Dentistry & Biomedical Sciences
  Research Group: Centre for Cancer Research and Cell Biology
  Dr E Kerr, Dr I Mills
Application Deadline: 30 January 2019
Mutant KRAS driven metabolic reprogramming is a key hallmark of cancer, providing the building blocks necessary to support aberrant cancer cell proliferation and is often achieved through rewiring glycolytic, mitochondrial and fatty acid metabolism, reportedly leading to mitochondrial dysfunction.
Details
Type/Funding
  Metabolic rewiring in liver cancer: Role of oxidative stress and the Nrf2 pathway
Peter MacCallum Cancer Centre Sir Peter MacCallum Department of Oncology, University of Melbourne
  Dr A. Cox
Applications accepted all year round
Many of the major risks factors for developing liver cancer such as alcohol, obesity, smoking and toxin exposure share in common a role for oxidative stress.
Details
Type/Funding
  Mybl2 in the regulation of cell fate decisions
University of Birmingham Institute of Cancer and Genomic Sciences
  Dr P Garcia
Applications accepted all year round
We are interested in the molecular mechanisms regulating cell fate decisions that occur during normal development, somatic reprogramming and cancer.
Details
Type/Funding
  Epitranscriptomic mechanisms in the maternal to zygotic transition of vertebrate embryos
University of Birmingham School of Biosciences
  Research Group: BBSRC MIBTP
  Dr M Soller, Prof F Mueller
Applications accepted all year round
Most mRNAs contain modified nucleotides in mRNA, but we know very little about their function (Haussmann et al, 2016, Dezi et al, 2016, Roignant and Soller, 2017).
Details
Type/Funding
  Modelling the interface of metabolism, methylation and mitochondria in prostate cancer
University of St Andrews School of Mathematics and Statistics
  Prof A Lynch
Applications accepted all year round
The interface of metabolism, methylation and mitochondria is of key importance for understanding the aetiology, biology and prognosis of prostate cancer[1].
Details
Type/Funding
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