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tau PhD Projects, Programs & Scholarships

We have 8 tau PhD Projects, Programs & Scholarships

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  Investigations into the neuroprotective actions of leptin in models of tau-related synaptic dysfunction.
  Dr J Harvey, Dr G H Doherty
Application Deadline: 14 January 2019
Key pathological features of AD are accumulation of amyloid plaques and neurofibrillary tangles comprising hyper-phosphorylated tau.
  Exploring the pathogenic roles of Rab GTPases in models of taupathy
  Prof F Giorgini, Prof C P Kyriacou
Application Deadline: 28 January 2019
Alzheimer’s disease (AD) is the most common neurodegenerative disorder and the predominant cause of dementia. The main pathological hallmarks of the disease in the brain are the presence of amyloid plaques and neurofibrilliary tangles composed of hyperphosphorylated forms of tau (P-tau).
  Examining the long-term effects of a concussive impact on accumulation of abnormal tau
  Dr F Corrigan
Applications accepted all year round
Applications are invited to undertake a PhD/Higher Degree by Research (HDR) to investigate how concussive impacts can lead to later neurodegenerative disease.
  Investigations into the neuroprotective actions of leptin and leptin-based mimetics in models of tauopathy.
  Dr J Harvey
Applications accepted all year round
In neurodegenerative disorders like Alzheimer’s disease (AD), synaptic communication within brain networks are impaired leading to significant memory deficits and dementia.
  Impact of 14-3-3 protein on the formation of protein aggregates involved in neurodegenerative diseases
  Dr J Hritz, Prof V Bryja, Dr J Pribyl
Application Deadline: 3 February 2019
The neuronal cells of patients suffering from neurodegenerative diseases are characteristic by possession of aggregates of several proteins such as tau and Abeta protein.
  Structural changes of the selected protein fibrils induced by phosphorylation and the interaction with 14-3-3 proteins
  Dr J Hritz, Prof V Bryja
Application Deadline: 15 March 2019
Several neurodegenerative diseases are asociated with the formation of fibrous (fibrillar) protein agreggates. The fibrillization of amyloid beta peptide into amyloid plaques and the agregation of hyperphosphorylated tau protein into neurofibrillar tangles are main neuropatological signs of Alzheimer disease.
  MRC DTP 4 Year PhD Programme: Investigations into the neuroprotective actions of leptin in cellular models of tauopathy
  Dr J Harvey, Dr M Stavridis
Application Deadline: 18 January 2019
This project is part of our exciting and challenging University of Dundee 4-year MRC DTP Programme in Quantitative and Interdisciplinary approaches to biomedical science.
  Inhibiting amyloid development using natural compounds: a molecular dynamics study
  Dr P Mulheran, Dr V A Ferro
Applications accepted all year round
Amyloid fibrils are highly ordered protein aggregates associated with many neurodegenerative diseases such as Alzheimer’s. Flavanoids, which are polyphenols such as curcumin present in the human diet, have been shown to inhibit fibril formation in tau protein which is abundant in neurons.
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