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muscular dystrophy PhD Projects, Programs & Scholarships

We have 10 muscular dystrophy PhD Projects, Programs & Scholarships

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  Small molecule probes to study human enzymes involved in muscular dystrophy
  Dr L Willems, Dr M A Fascione
Application Deadline: 8 January 2020

Funding Type

PhD Type

Background. Proteins on the surface of cells are often modified with diverse, complex carbohydrate structures. These carbohydrates, named glycans, play central roles in biology by regulating, for instance, cell recognition and signalling events.
  Novel therapeutic approaches for the treatment of Duchenne muscular dystrophy.
  Prof S Winder
Applications accepted all year round

Funding Type

PhD Type

Duchenne muscular dystrophy (DMD) is caused by mutations in the DMD gene leading to loss of the cytoskeletal protein dystrophin.
  Gene and Cell Therapy For Muscular Dystrophy, Muscular Atrophy, Diabetes and Metabolic conditions
  Dr K Foster
Applications accepted all year round

Funding Type

PhD Type

Skeletal muscle development is a complex process and a number of regulatory factors for the different steps involved in stem cell activation and proliferation through to multinucleated, post-mitotic cells have been identified.
  Statistical modelling of disease progression in Spinal Muscular Atrophy and Duchenne Muscular Dystrophy
  Dr G Baranello, Prof F Muntoni, Dr D Ridout
Application Deadline: 29 November 2019

Funding Type

PhD Type

A 3-year PhD Studentship is available within the Dubowitz Neuromuscular Centre, UCL GOS Institute of Child Health under the supervision of Professor Francesco Muntoni, Dr Giovanni Baranello and Ms Deborah Ridout.
  Development of gene therapies for muscular dystrophies
  Dr L Popplewell
Applications accepted all year round

Funding Type

PhD Type

The activities in the Popplewell lab focus on the development of pre-clinical gene therapies for neuromuscular diseases, in particular muscular dystrophies based on gene replacement, endonuclease-mediated gene editing, antisense oligonucleotide (AO)-induced exon skipping and gene sliencing.
  Structure and activity of carbohydrate-active enzymes associated with human disease
  Dr L Willems, Prof G Davies
Application Deadline: 1 February 2020

Funding Type

PhD Type

Proteins on the surface of mammalian cells are often modified with diverse, complex carbohydrate structures. These carbohydrates, named glycans, play key roles in a range of cellular processes including cell-cell and host-pathogen interactions.
  Structure and activity of carbohydrate-active enzymes associated with human disease
  Dr L Willems, Prof G Davies
Application Deadline: 1 February 2020

Funding Type

PhD Type

Proteins on the surface of mammalian cells are often modified with diverse, complex carbohydrate structures. These carbohydrates, named glycans, play key roles in a range of cellular processes including cell-cell and host-pathogen interactions.
  Tackling Rare Neuromuscular Diseases
  Dr Y Jamshidi, Dr L Southgate, Dr D Osborn
Application Deadline: 1 January 2020

Funding Type

PhD Type

Limb-girdle muscular dystrophies (LGMD’s) comprise 30% of all progressive muscular dystrophies, collectively affecting up to 500,000 individuals worldwide.
  (BBSRC DTP) The role of collagen VI in the skeletal muscle stem cell niche using combined imaging approaches including Cryo-EM
  Prof C Baldock, Prof G Cossu, Dr A Roseman
Application Deadline: 31 January 2020

Funding Type

PhD Type

Collagen VI is a unique member of the collagen superfamily which has a short collagenous region but arrays of globular domains and assembles to form beaded microfibrils.
  Precision Medicine DTP - 3D Modeling of Changes in Genome Architecture in Development and Disease
  Research Group: Institute of Cell Biology
  Prof E Schirmer, Dr D Marenduzzo
Application Deadline: 8 January 2020

Funding Type

PhD Type

Background. Defects in spatial genome organisation have recently been linked to human disease and the National Institutes of Health, USA director speculated that most as yet unidentified disease alleles will occur in non-coding genome regions.
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