Our group studies mechanisms of brain health, ageing and disease focusing on the lysosomal protein transmembrane protein 106B (TMEM106B) which forms amyloids in the brain in an age-dependent manner. Read more
CD4+Foxp3+ regulatory T-cells (Tregs) are the most well characterised suppressive cell type in the immune system, with potent roles in suppressing autoimmunity. Read more
Dementia encompasses various subtypes such as Alzheimer's disease, vascular dementia, and frontotemporal dementia, each affecting different aspect of brain structure and function. Read more
Supervisor. Prof. Rita Horvath . Co-supervisor: Dr. Daniel Lagos . Mitochondria are specialised cell organelles which transform nutrients into energy. Read more
To improve our understanding of the progression of neurodegenerative disease, and the mechanisms involved, we need to combine different types of neuroimaging, and integrate it with clinical and genetic data. Read more
With strong alignment to the clinical programmes within the Department of Clinical Neurosciences and other departments in the Clinical School, our programme… Read more
The project focuses on the intracellular stages of amyloidogenesis, aiming to unravel the complex mechanisms contributing to diseases such as Alzheimer's. Read more
Our aim is to develop novel Positron Emission Tomography (PET) radiotracers for imaging the brain. PET is a non-invasive molecular imaging technique which enables visualization and quantification of various biological targets in vivo (e.g., receptor, enzyme). Read more
Neuroinflammation is an important driving mechanism in all diseases caused by frontotemporal lobar degeneration, and predicts clinical progression. Read more
Multiple sclerosis (MS) is a devastating neurological condition affecting millions worldwide. Cognitive impairment significantly impacts the quality of life of people with MS and is often irreversible. Read more
Mitochondrial diseases are caused by defects in genes required for energy production and oxidative phosphorylation (OxPhos). We find it intriguing that some patients with mitochondrial disease present late in life, with very tissue-specific phenotypes. Read more
It is now well established that both gastrointestinal dysfunction and immune activation occur in early Parkinson’s disease, and emerging evidence indicates that these processes are linked to faster disease progression. Read more
*Offer only available for the duration of your active subscription, and subject to change. You MUST claim your prize within 72 hours, if not we will redraw.
Do you want hassle-free information and advice?
Create your FindAPhD account and sign up to our newsletter:
Find out about funding opportunities and application tips
Receive weekly advice, student stories and the latest PhD news
Hear about our upcoming study fairs
Save your favourite projects, track enquiries and get personalised subject updates
Due to your Facebook privacy settings, we were unable to create your account at this time. Please select another method to sign up.
We were unable to log you in with your Google account at this time. If you have third-party cookies blocked, please enable them, refresh, and try again.
or
Continue with Facebook
Create your account
We were unable to log you in with your Google account at this time. If you have third-party cookies blocked, please enable them, refresh, and try again.
Looking to list your PhD opportunities? Log in here.