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Cell Biology / Development (mobility) PhD Projects, Programs & Scholarships

We have 10 Cell Biology / Development (mobility) PhD Projects, Programs & Scholarships

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  Role of pentraxin and interaction with complement in immune defence against opportunistic infections
  Prof C Garlanda, Prof S Meri
Application Deadline: 10 November 2019

Funding Type

PhD Type

PTX3 is a soluble pattern recognition molecule which acts as a key component of humoral innate immunity in opportunistic infections of fungal and bacterial origin.
  Identification of novel complement evasion mechanisms developed by bacterial pathogens
  Prof A Blom, Prof S Rooijakkers
Application Deadline: 10 November 2019

Funding Type

PhD Type

The project is focused on discovery of new complement evasion mechanisms developed by bacterial pathogens. One of the studied bacteria will be opportunistic Filifactor alocis, a recently identified periodontal pathogen of major importance but still poorly studied.
  Development of immunoassays for specific classical and lectin pathway activation markers
  Prof Z Prohaszka, Prof R Würzner
Application Deadline: 10 November 2019

Funding Type

PhD Type

Infections and various immune-inflammation mediated conditions are characterized by the activation of the complement system. Antibodies (immune complexes) activate the classical pathway whereas repetitive carbohydrate structures give rise to the initiation of the lectin pathway.
  FH, FHR and PTX-3 binding to opportunistic bacteria and malaria parasites
  Prof S Meri, Prof C Garlanda
Application Deadline: 10 November 2019

Funding Type

PhD Type

Infections pose a global threat because of spread of antibiotic resistance, hospital infections and problems in the third world. On the other hand, rapidly increasing information of microbial genomes now provides new opportunities to tackle virulence mechanisms of pathogenic microbes.
  Characterization of C5aR2 expression and function in Toxoplasma gondii infection
  Prof J Koehl, Prof D Wilflingseder
Application Deadline: 10 November 2019

Funding Type

PhD Type

Toxoplasma gondii (T. gondii) is a widespread obligate intracellular protozoan parasite. It is of major medical importance during pregnancy and in immunocompromised individuals.
  PhD student (m/f/d) in Systems Biology of Epigenetic Regulation
  Dr E Schulz
Applications accepted all year round

Funding Type

PhD Type

The Max Planck Institute for Molecular Genetics is an international research institute with approximately 350 employees, working in the field of genomics and gene regulation.
  Application of molecular methods for the discovery of anticancer agents
  Prof S Cosconati
Application Deadline: 4 July 2020

Funding Type

PhD Type

The PhD program in Molecular Life Sciences is a research doctorate of the University of Campania Luigi Vanvitelli that is run with the collaboration of the Italian National Research Council (CNR).
  Single and double inhibition of complement and CD14 in opportunistic conditions
  Prof T Mollnes, Prof R Würzner, Prof P Garred
Application Deadline: 10 November 2019

Funding Type

PhD Type

The group of professor Mollnes has worked with a combined inhibition of complement (at the level of C3 and C5), and the Toll like receptors (TLRs) targeting CD14, a key co-receptor for TLR4, TLR2 and others, based on an hypothesis to attenuating the upstream innate immune activation when it is over- or dys-activated.
  Influence of HIV-1 opsonization on APC functions with regard to persistence of the virus and opportunistic pathogens, such as Mycobacteria spp. within relevant human 3D models
  Prof D Wilflingseder, Prof S Niemann
Application Deadline: 10 November 2019

Funding Type

PhD Type

HIV-1 and Mycobacterium tuberculosis represent detrimental co-epidemics worldwide, particularly in sub-Saharan Africa, and co-infection accelerates progression of both diseases.
  Using CRISPR in iPS cells to modify platelet function
  Dr C.E. Hughes
Applications accepted all year round

Funding Type

PhD Type

"Platelets are the small cells in the blood whose job it is to prevent bleeding. Under normal conditions, when they encounter a damaged blood vessel they become activated and form a thrombus.
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