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Endocrinology (self) PhD Projects, Programs & Scholarships

We have 33 Endocrinology (self) PhD Projects, Programs & Scholarships

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  Investigating metabolic changes in neurodegeneration
  Dr F Mackenzie, Dr M Stolinski
Applications accepted all year round

Funding Type

PhD Type

This 3-year PhD will study the increasingly recognised association between metabolic disease and neurodegeneration using in vitro cell culture methods.
  Investigating fatigue and the response to exercise training in clinical population with functional magnetic resonance imaging (fMRI) and magnetic resonance spectroscopy (MRS)
  Dr H Kubis
Application Deadline: 24 January 2020

Funding Type

PhD Type

PhD Studentship/Junior Technician. Applications are invited for a six year, 50% FTE part time PhD studentship/50% FTE part time Junior Technician post funded by the School of Sport, Health and Exercise Sciences at Bangor University.
  Interrogating the role of retriever sorting complex in adipose insulin-sensitivity and GLUT4 recycling
  Dr P Whitley
Applications accepted all year round

Funding Type

PhD Type

The University of Bath is inviting applications from self-funded applicants for the following PhD opportunity. Supervisors. Dr Paul Whitley, Department of Biology & Biochemistry https://researchportal.bath.ac.uk/en/persons/paul-whitley.
  Epidemiology of common endocrine disorders and diabetes
  Dr T Sathyapalan
Applications accepted all year round

Funding Type

PhD Type

A PhD project is available in Prof Sathyapalan group with a focus on the epidemiology of common endocrine disorders and diabetes at the University of Hull.
  MHC class I antigen presentation in autoimmune diabetes mellitus
  Prof M Garstka
Application Deadline: 9 February 2020

Funding Type

PhD Type

A Ph.D. program starting in September 2020 is now open for the applications. The Ph.D. student will work with Prof. Malgorzata Garstka at Core Research Laboratory, the Second Affiliated Hospital, Medical School of Xi’an Jiaotong University, Xi’an, China.
  Ion channel signalling in cancer cells
  Dr W Brackenbury
Application Deadline: 30 June 2020

Funding Type

PhD Type

Our cells constantly sense and transport ions present in their environment. From embryonic development to epilepsy to heart disease to cancer, our cells’ ability to respond to changes in the ionic microenvironment is essential for healthy ageing.
  Role of telomere attrition in cardiometabolic disease risk
  Dr J Buxton, Dr N Hill
Applications accepted all year round

Funding Type

PhD Type

Common conditions such as Type 2 diabetes (T2D) and coronary artery disease (CAD) are complex disorders caused by a myriad of interacting genetic and lifestyle factors.
  The role of matrix interactions in regulating the SPARC family of matricellular proteins.
  Dr N Hill, Dr A Munasinghe
Applications accepted all year round

Funding Type

PhD Type

The SPARC family of matricellular proteins determine the way in which cells respond to their extracellular environment. SPARC family proteins interact with a range of binding partners, including matrix proteins (1).
  Building the human endometrium in vitro: The role of macrophages in receptive and decidual endometrium to optimise reproductive health
  Dr E Mann, Dr Peter Ruane, Prof J Aplin, Dr L Mohiyiddeen
Applications accepted all year round

Funding Type

PhD Type

The human endometrium is a highly dynamic tissue that rapidly generates over ~20 days and differentiates to form a uterus lining receptive to embryo implantation, further transforming into decidua to support embryonic development, or breaking down before regenerating a few days later.
  Cellular protein quality control in diabetes-associated heart disease: The insight into mechanisms and therapeutic potentials
  Dr V Liu, Dr L Swanton
Applications accepted all year round

Funding Type

PhD Type

Cardiovascular complications are the leading causes of diabetes mortality. With the exception of vascular and valvular injuries, diabetic cardiomyopathy (DCM) is a distinct myocardial disease, which is characterised by abnormal cellular metabolism and defects in organelles function, leading to impaired cardiac function.
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