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Cell Biology / Development PhD Projects, Programs & Scholarships in Liverpool

We have 33 Cell Biology / Development PhD Projects, Programs & Scholarships in Liverpool

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Showing 1 to 15 of 33
  MRC DiMeN Doctoral Training Partnership: Targeting lung fibrosis: Dissecting molecular crosstalk mechanisms that drive disease pathogenesis and therapeutic response
  Dr M Morgan, Prof L Yu
Application Deadline: 6 January 2020

Funding Type

PhD Type

Idiopathic pulmonary fibrosis (IPF) is a chronic, progressive, lung disease associated with high morbidity and median survival of.
  Mechanistic mathematical/computational modelling of the PAR polarity network
  Dr N Savage, Dr J Rodriguez
Applications accepted all year round

Funding Type

PhD Type

Cell function is dependent on cell shape. For example, dendritic spine morphology changes from filopodia, which are thought to be searching structures, to mushroom shaped once a potential synaptic connection is found, increasing the postsynaptic surface.
  The Biology of Parasitism in Parasitic Nematodes
  Prof M Viney
Applications accepted all year round

Funding Type

PhD Type

We work on the biology of parasitic nematode worms, particularly Strongyloides spp. The recent, detailed characterisation of the Strongyloides genome (Hunt et al.
  The Eco-Immunology of Wild Mice
  Prof M Viney
Applications accepted all year round

Funding Type

PhD Type

The immune responses that wild animals make contribute to their fitness, but what those immune responses are – and how they differ from those of laboratory animals – is hardly known.
  Identification of blood biomarkers for antenatal diagnosis of a common congenital heart defect, Tetralogy of Fallot.
  Dr I Dykes
Application Deadline: 1 December 2019

Funding Type

PhD Type

Almost 1 in every 100 babies born in the UK has a congenital heart defect (CHD). CHD is the leading cause of infant mortality and affected children often require surgery at birth.
  MRC DiMeN Doctoral Training Partnership: Understanding the mechanism of actin-microtubule co-regulation underlying neurodegeneration in motor neurons
  Dr T Zech, Dr A Twelvetrees
Application Deadline: 6 January 2020

Funding Type

PhD Type

Disrupted microtubule dynamics are a key mechanism underlying the selective vulnerability of neurons across many different motor neuron diseases.
  MRC DiMeN Doctoral Training Partnership: How does a common SNP predispose to age-related cardiovascular and musculoskeletal disease? A tissue engineering and lncRNA approach using MSCs
  Dr EG Laird, Dr JR Henstock, Dr D Young, Prof K Stellos
Application Deadline: 6 January 2020

Funding Type

PhD Type

IMPORTANCE. A single nucleotide polymorphism (SNP) present in one quarter of the population (rs1800012) is associated with osteoporotic fracture, intervertebral disc degeneration and cardiovascular disease1-3.
  MRC DiMeN Doctoral Training Partnership: Targeting cancer through disruption of the ubiquitin economy
  Prof M Clague, Prof S Urbé
Application Deadline: 6 January 2020

Funding Type

PhD Type

A PhD project in the shared laboratory of Prof. Michael Clague and Prof. Sylvie Urbé http://pcwww.liv.ac.uk/~clague/ in collaboration with an industry partner.
  MRC DiMeN Doctoral Training Partnership: Functional and genetic analyses of HSPB mutations underlying progressive neuronal deficits in Charcot-Marie-Tooth disease
  Dr J Barclay, Dr E Seward, Dr D Hammond
Application Deadline: 6 January 2020

Funding Type

PhD Type

Understanding the cellular and molecular mechanisms underlying progressive neurological deterioration is a key challenge in modern biomedical research.
  MRC DiMeN Doctoral Training Partnership: Developing Novel Bispecific Antibody-Drug Conjugates to Treat Leukaemia
  Dr J Woolley, Dr J Slupsky
Application Deadline: 6 January 2020

Funding Type

PhD Type

Acute myeloid leukaemia (AML) is the second most common leukaemia, accounting for 32% of all leukaemia in diagnosed in adults.
  MRC DiMeN Doctoral Training Partnership: Liquid biopsy of the liver to improve molecular targeted cancer therapy
  Prof C Goldring, Prof F Falciani, Dr A Chadwick, Dr M Cross
Application Deadline: 6 January 2020

Funding Type

PhD Type

Molecular targeted therapies have revolutionised oncology. However, whilst patient life-expectancy is improving, these therapies carry a risk of causing damage to healthy tissues in the body, especially to the liver.
  MRC DiMeN Doctoral Training Partnership: Combining single-cell proteomics and RNA sequencing to track heterogeneity in alphavirus infection
  Dr E Emmott, Dr A Darby
Application Deadline: 6 January 2020

Funding Type

PhD Type

Arboviruses spread from person to person through an insect vector. They include the alphaviruses (e.g. chikungunya virus –CHIKV, and the more tractable BSL2 model O’Nyong-Nyong virus - ONNV) which are endemic in Africa and cause significant morbidity and potentially life-long complications.
  MRC DiMeN Doctoral Training Partnership: Investigating the potential of a regenerative medicine therapy to treat cardiorenal disease
  Prof P Murray, Prof J Schneider, Prof H Poptani, Dr B Wilm
Application Deadline: 6 January 2020

Funding Type

PhD Type

Cardiovascular disease (CVD) is the leading cause of mortality in patients with chronic kidney disease (CKD). The strong association between CKD and CVD has been recognised for many years, but the mechanisms by which CKD causes CVD are largely unknown.
  Does phenotypic plasticity help or hinder rapid adaptation?
  Dr S J Plaistow, Prof J Hill, Dr V Oostra
Application Deadline: 8 January 2020

Funding Type

PhD Type

Anthropogenic stress is altering our planet at an unprecedented rate and has major implications for global biodiversity, health and the economy.
  The effect of low protein diet on the development of the musculoskeletal system
  Dr A Vasilaki, Prof MJ Jackson, Dr I Kanakis, Dr K Whysall
Applications accepted all year round

Funding Type

PhD Type

Age-related sarcopenia is characterised by reduction in the number of myofibres and motor neurons and an additional weakening of the remaining fibres, causing reduction in muscle mass and function.
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