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Neuroscience / Neurology (human) PhD Projects, Programs & Scholarships

We have 93 Neuroscience / Neurology (human) PhD Projects, Programs & Scholarships

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  Regulation of autophagy in human embryonic stem cells and its therapeutic application in induced pluripotent stem cell-based disease models
  Dr S Sarkar
Applications accepted all year round

Funding Type

PhD Type

Project background. Regulation of proteostasis is critical for maintaining tissue homeostasis. Autophagy, a major intracellular degradation pathway essential for cellular and energy homeostasis, functions in the clearance of aggregation-prone proteins and damaged organelles.
  (MRC DTP) Developing a zebrafish model for a novel human neurodevelopmental disorder
  Prof G D Pavitt, Dr P Kasher, Dr S Banka
Application Deadline: 15 November 2019

Funding Type

PhD Type

Background. We have recently discovered a novel human disorder caused by mutations in the EIF5A1 gene resulting in developmental problems, small head size and craniofacial defects in children [1].
  A 3D in vitro model of wound healing incorporating skin, nerves and immune cells
  Dr A M Rajnicek, Dr H M Wilson
Applications accepted all year round

Funding Type

PhD Type

Diabetes, paralysis, being bed ridden and other conditions can result in chronic wounds that fail to heal, with significant personal consequences for patients and an annual cost of £4.5-5.1 billion annually (Guest et al., 2015) to the National Health Service.
  Developing nutritional guidelines for human breast milk bank donors
  Dr S Dyall, Dr Y Jeanes
Applications accepted all year round

Funding Type

PhD Type

Appropriate nutrition is particularly important for preterm infants, since their maternal nutrient supply has been prematurely interrupted.
  (MRC DTP) RAC1 mutations in human neurodevelopmental syndrome: from mechanism to treatment
  Dr T Millard, Dr S Banka, Dr S Woolner, Prof V Allan
Application Deadline: 15 November 2019

Funding Type

PhD Type

RAC1 regulates a variety of essential cellular functions1,2,3. We recently showed that it is critical for normal human development by discovering a novel neurodevelopmental syndrome caused by mutations in the RAC1 gene4.
  Exploring the mitotic functions of ASPM in human brain size regulation
  Dr J Bond, Dr E E Morrison, Prof M Peckham
Applications accepted all year round

Funding Type

PhD Type

The increase in relative brain size is one of the most striking events in human evolution. To determine how human brain size is normally regulated we have investigated the cause of autosomal recessive primary microcephaly (MCPH), a congenital disorder of reduced brain size and associated mental retardation.
  Auditory cognition and human factors
  Dr P Beaman
Applications accepted all year round

Funding Type

PhD Type

Expressions of interest are invited on any topics related to either auditory cognition and working memory or to human factors as they relate to energy conservation and climate change.
  Developing a therapy for hearing loss using human pluripotent stem cells
  Prof M Rivolta
Applications accepted all year round

Funding Type

PhD Type

The vast majority of cases of deafness are produced by the irreversible loss of the sensory hair cells and their associated neurons.
  To treat or not to treat: investigating the impact of depression and antidepressant use in pregnancy.
  Prof RM John
Application Deadline: 25 November 2019

Funding Type

PhD Type

Depression in pregnancy, often occurring in combination with anxiety and a stressful environment, increases the risk of adverse outcomes for offspring with males showing more attention deficits, cognitive problems and externalising behaviour and females tending to present with anxiety, depression and internalising symptoms.
  (MRC DTP) Understanding the mechanism and finding treatment of neurodevelopmental disorder caused by KMT2C mutations
  Dr S Banka, Prof S J Kimber
Application Deadline: 15 November 2019

Funding Type

PhD Type

Background. We recently identified a novel human syndrome caused by mutations in a gene called KMT2C1. Patients with this condition predominantly have developmental delay and intellectual disability indicating the important function of this gene in brain’s normal development and function.
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