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Neuroscience / Neurology (screening) PhD Projects, Programs & Scholarships

We have 13 Neuroscience / Neurology (screening) PhD Projects, Programs & Scholarships

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  Genome and transcriptome sequencing and functional analysis to find new mutation types in patients with inherited blindness
  Prof C Inglehearn
Applications accepted all year round

Funding Type

PhD Type

Human inherited retinal dystrophies (IRDs) result from mutations in over 200 different genes, many of them first implicated by the Leeds Vision Research Group (eg Panagiotou E et al 2017, AJHG 100:960-968; El-Asrag M et al 2015, 96:948-54).
  SWBio DTP PhD project: Amyloid Transcription Block Survival - Deriving Functionally Active Peptide Inhibitors of Amyloidosis and Toxicity
  Prof J Mason
Application Deadline: 2 December 2019

Funding Type

PhD Type

This project is one of a number that are in competition for funding from the South West Biosciences Doctoral Training Partnership (SWBio DTP).
  Retinal vessel size and shape associations with brain structure and function
  Prof AR Rudnicka, Dr C Frost
Application Deadline: 1 January 2020

Funding Type

PhD Type

The size and shape of retinal vessels might be early markers of decline in cognitive function and dementia. We have used novel artificial intelligence enhanced automated software to generate a rich quantitative characterization of retinal vessels in UK Biobank participants who underwent retinal imaging.
  Using zebrafish models to investigate cerebral arteriovenous malformations
  Dr P Kasher, Prof S Allan, Dr A Parry-Jones, Dr H Patel
Applications accepted all year round

Funding Type

PhD Type

Cerebral Arteriovenous Malformations (cAVM) relate to a spectrum of conditions associated with blood vessel abnormalities of the brain [1].
  Regulation of autophagy in human embryonic stem cells and its therapeutic application in induced pluripotent stem cell-based disease models
  Dr S Sarkar
Applications accepted all year round

Funding Type

PhD Type

Project background. Regulation of proteostasis is critical for maintaining tissue homeostasis. Autophagy, a major intracellular degradation pathway essential for cellular and energy homeostasis, functions in the clearance of aggregation-prone proteins and damaged organelles.
  Finding the eat-me signals
  Dr I Ganley
Application Deadline: 1 December 2019

Funding Type

PhD Type

The Ganley lab is interested in unravelling the molecular mechanism of autophagy (which literally translates from the Greek meaning to eat oneself).
  China Scholarship Council - Finding the eat-me signals
  Dr I Ganley
Application Deadline: 15 January 2020

Funding Type

PhD Type

The School of Life Sciences at the University of Dundee, joint with the China Scholarship Council (CSC), is proud to be able to offer a scholarship programme for postgraduate research students.
  P2X receptors in the peripheral nervous system: molecular pharmacology (FOUNTAINSU20SF)
  Dr S Fountain
Application Deadline: 31 May 2020

Funding Type

PhD Type

P2X receptors are ligand-gated ion channels activated by extracellular adenosine 5’-triphosphate (ATP). In the peripheral nervous system (PNS), ATP is a neurotransmitter acting in both autonomic and sensory nerve control of body tissue function and homeostasis.
  The influence of JAKMIP1, a key risk gene for autism, on neural circuit function. PhD in Medical Studies (MRC GW4 BioMed DTP)
  Dr A Oguro-Ando
Application Deadline: 25 November 2019

Funding Type

PhD Type

Supervisory team. Dr Asami Oguro-Ando, The Institute of Biomedical and Clinical Science, College of Medicine and Health, University of Exeter.
  Understanding amyloid aggregation mechanisms of alpha- and gamma synuclein in vitro and in vivo
  Research Group: BBSRC White Rose DTP
  Dr P van Oosten-Hawle, Prof S E Radford
Application Deadline: 6 January 2020

Funding Type

PhD Type

A key pathological hallmark of synucleopathies, including Parkinson’s Disease (PD) and Amyotrophic Lateral Sclerosis (ALS), is the formation of cytotoxic alpha-synuclein and gamma-synuclein aggregates respectively, that lead to neuronal cell death.
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