ARUK provide a student stipend of £17,000 per annum plus tuition fees at Home/EU rate and research/travel costs.
Studentship is open to UK/EU students only.
English language criteria apply.
Applicants are required to hold a first or upper-second class UK Bachelor’s degree in an appropriate subject, or a recognised Master’s degree.
DeJesus-Hernandez, M., et al., Expanded GGGGCC hexanucleotide repeat in noncoding region of C9ORF72 causes chromosome 9p-linked FTD and ALS. Neuron, 2011. 72(2): p. 245- 56.
Renton, A.E., et al., A hexanucleotide repeat expansion in C9ORF72 is the cause of chromosome 9p21-linked ALS-FTD. Neuron, 2011. 72(2): p. 257-68.
Moens, T.G., L. Partridge, and A.M. Isaacs, Genetic models of C9orf72: what is toxic? Curr Opin Genet Dev, 2017. 44: p. 92-101.
Balendra, R. and A.M. Isaacs, C9orf72-mediated ALS and FTD: multiple pathways to disease. Nat Rev Neurol, 2018. 14(9): p. 544-558.
Mori, K., et al., The C9orf72 GGGGCC repeat is translated into aggregating dipeptide-repeat proteins in FTLD/ALS. Science, 2013. 339(6125): p. 1335-8.
Ash, P.E., et al., Unconventional translation of C9ORF72 GGGGCC expansion generates insoluble polypeptides specific to c9FTD/ALS. Neuron, 2013. 77(4): p. 639-46.
Mizielinska, S., et al., C9orf72 repeat expansions cause neurodegeneration in Drosophila through arginine-rich proteins. Science, 2014. 345(6201): p. 1192-1194.
Alic, N., et al., Genome-wide dFOXO targets and topology of the transcriptomic response to stress and insulin signalling. Mol Syst Biol, 2011. 7: p. 502.
Castillo-Quan, J.I., et al., Lithium Promotes Longevity through GSK3/NRF2-Dependent Hormesis. Cell Rep, 2016. 15(3): p. 638-650.
FTE Category A staff submitted: 172.90
Research output data provided by the Research Excellence Framework (REF)Click here to see the results for all UK universities