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Virology (cell culture) PhD Projects, Programs & Scholarships

We have 14 Virology (cell culture) PhD Projects, Programs & Scholarships

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  Characterising protective local and systemic immune responses to Group A streptococcal throat infection in children
  Prof A Finn, Dr L B Nicholson
Application Deadline: 25 November 2019

Funding Type

PhD Type

The project. Group A streptococcus (GAS) causes infective, invasive and post-infective immune-mediated diseases including serious heart and kidney conditions resulting in significant illness and 0.5-1.5 million deaths/year globally.
  MRC DiMeN Doctoral Training Partnership: Combining single-cell proteomics and RNA sequencing to track heterogeneity in alphavirus infection
  Dr E Emmott, Dr A Darby
Application Deadline: 6 January 2020

Funding Type

PhD Type

Arboviruses spread from person to person through an insect vector. They include the alphaviruses (e.g. chikungunya virus –CHIKV, and the more tractable BSL2 model O’Nyong-Nyong virus - ONNV) which are endemic in Africa and cause significant morbidity and potentially life-long complications.
  Virus manipulation of host cell micro-RNAs to regulate gene expression
  Research Group: BBSRC White Rose DTP
  Dr A MacDonald, Prof A Whitehouse
Application Deadline: 6 January 2020

Funding Type

PhD Type

micro-RNAs (miRNAs) are abundant endogenous RNAs and emerging evidence suggests they regulate gene expression by multiple processes.
  Precision Medicine DTP - How do IFITM proteins regulate the Interferon Related DNA-damage Resistance Signature (IRDS) and clinical outcomes in cancers of high unmet clinical need?
  Dr R Sloan, Prof K Ball, Dr S Wilson
Application Deadline: 8 January 2020

Funding Type

PhD Type

Background. Interferon Induced Transmembrane proteins (IFITMs) are a family of innate immune membrane proteins that can inhibit viral infection at cell entry (e.g.
  PhD student position is available to work on extracellular vesicles in cardiovascular biology
  Dr X Osteikoetxea
Application Deadline: 15 December 2019

Funding Type

PhD Type

PhD student position is available to work on extracellular vesicles in cardiovascular biology. The position will be based in Semmelweis University, Budapest, Hungary.
  Control and inhibition of virus replication
  Research Group: Astbury Centre for Structural Molecular Biology
  Dr A.K Tuplin
Applications accepted all year round

Funding Type

PhD Type


The Tuplin laboratory utilises a range of cutting-edge approaches to investigate how arboviruses - specificaly Chikungunya, Dengue and Zika viruses - control replication and translation of their genomes through interactions between RNA structures, host cell proteins and non-coding RNA, and the potential of such RNA elements/interactions as novel therapeutic targets.
  Synthetic Virology: Development of gene delivery vectors/synthetic vaccines
  Research Group: Astbury Centre for Structural Molecular Biology
  Prof P G Stockley
Applications accepted all year round

Funding Type

PhD Type

There is worldwide interest in exploiting our modern understanding of genomics to target gene expression therapeutically via a variety of mechanisms, such as transgene insertion and CRISPR-Cas gene editing.
  A newly discovered protein encoded by enteroviruses: from mechanism to application
  Dr A Firth, Dr V Lulla
Application Deadline: 1 January 2020

Funding Type

PhD Type

Enteroviruses have single-stranded RNA genomes of ~7.4 kb.
  Development of gene therapies for muscular dystrophies
  Dr L Popplewell
Applications accepted all year round

Funding Type

PhD Type

The activities in the Popplewell lab focus on the development of pre-clinical gene therapies for neuromuscular diseases, in particular muscular dystrophies based on gene replacement, endonuclease-mediated gene editing, antisense oligonucleotide (AO)-induced exon skipping and gene sliencing.
  EASTBIO: How do the HUSH complex and REAF/RPRD2 control endogenous retroelements?
  Dr R Sloan, Dr J Garcia-Perez
Application Deadline: 5 January 2020

Funding Type

PhD Type

The aim of this project is to understand how the recently identified antiviral HUSH complex and the antiviral protein REAF/RPRD2 can inhibit endogenous retroelements.
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