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Understanding how systemic infection exacerbates Alzheimer’s disease

  • Full or part time
  • Application Deadline
    Applications accepted all year round
  • Self-Funded PhD Students Only
    Self-Funded PhD Students Only

Project Description

Systemic infection and inflammation are associated with the risk and progression of Alzheimer’s disease but the underlying mechanisms are unclear. Our research indicates that systemic infection can rapidly and directly modulate key brain hallmarks of Alzheimer’s disease: amyloid plaques deposition and neurofibrillary tangles. The project aims to understand the role played by peripheral pro-inflammatory cytokines induced in response to immune stimulation in this effect. The work will use in vivo and ex vivo approaches. (e.g. behavioural, pharmacological and molecular) in a preclinical model of Alzheimer’s disease.

Funding Notes

Home applicants should contact the supervisor to determine the current funding status for this project. EU applicants should visit the Graduate School webpages for information on specific EU scholarships View Website. International applicants should visit our International Research Scholarships page for information regarding fees and funding at the University View Website.

How good is research at University of Nottingham in Biological Sciences?

FTE Category A staff submitted: 90.86

Research output data provided by the Research Excellence Framework (REF)

Click here to see the results for all UK universities

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